Search results for "Bleeding Time"

showing 4 items of 4 documents

Conformation-specific blockade of the integrin GPIIb/IIIa: a novel antiplatelet strategy that selectively targets activated platelets.

2006

Platelet activation causes conformational changes of integrin GPIIb/IIIa (α IIb β 3 ), resulting in the exposure of its ligand-binding pocket. This provides the unique possibility to design agents that specifically block activated platelets only. We used phage display of single-chain antibody (scFv) libraries in combination with several rounds of depletion/selection to obtain human scFvs that bind specifically to the activated conformation of GPIIb/IIIa. Functional evaluation of these scFv clones revealed that fibrinogen binding to human platelets and platelet aggregation can be effectively inhibited by activation-specific scFvs. In contrast to clinically used GPIIb/IIIa blockers, which ar…

Blood PlateletsCarotid Artery DiseasesBleeding TimePhysiologyAmino Acid MotifsMolecular ConformationEptifibatidePlatelet Glycoprotein GPIIb-IIIa ComplexFerric CompoundsAntibodiesMiceChloridesFibrinolytic AgentsmedicineAbciximabAnimalsHumansPlateletPlatelet activationChemistryFibrinogen bindingFibrinogenThrombosisTirofibanPlatelet ActivationMolecular biologyComplementarity Determining RegionsMice Inbred C57BLTirofibanImmunologyEptifibatidePlatelet aggregation inhibitorTyrosineCardiology and Cardiovascular MedicineGlycoprotein IIb/IIIaPeptidesPlatelet Aggregation Inhibitorsmedicine.drugCirculation research
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In vitro and in vivo characterization of a new organic nitrate hybrid drug covalently bound to pioglitazone.

2014

<b><i>Background/Aims:</i></b> Organic nitrates represent a group of nitrovasodilators that are clinically used for the treatment of ischemic heart disease. The new compound CLC-3000 is an aminoethyl nitrate (AEN) derivative of pioglitazone, a thiazolidinedione antidiabetic agent combining the peroxisome proliferator-activated receptor γ agonist activity of pioglitazone with the NO-donating activity of the nitrate moiety. <b><i>Methods:</i></b> In vitro and in vivo characterization was performed by isometric tension recording, platelet function, bleeding time and detection of oxidative stress. <b><i>Results:</i></…

DrugBlood PlateletsMaleBleeding TimePlatelet Aggregationmedia_common.quotation_subjectVasodilator Agentsmedicine.disease_causeMitochondria Heartchemistry.chemical_compoundNitrateFibrinolytic AgentsIn vivomedicineAnimalsHumansHypoglycemic AgentsRats WistarAortamedia_commonPharmacologyNitratesPioglitazoneChemistryGeneral MedicineReactive Nitrogen SpeciesIn vitroOrganic nitratesMice Inbred C57BLVasodilationBiochemistryCovalent bondVasoconstrictionThiazolidinedionesReactive Oxygen SpeciesPioglitazoneOxidative stressmedicine.drugPharmacology
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Aspirin treatment improves bladder function after outlet obstruction in rabbits.

2001

Abstract Objectives. To examine whether bladder smooth muscle dysfunction after outlet obstruction could be altered by treatment with aspirin. Long-term outlet obstruction causes contractile and metabolic dysfunction of the bladder in vivo and in vitro. The evidence is growing that a decrease in bladder perfusion is an important cause of this phenomenon. The platelet aggregation inhibitor, acetylsalicylic acid (aspirin), has been used to improve perfusion of the heart for decades. Methods. Ten male New Zealand white rabbits were obstructed for 4 weeks. Five rabbits received no further treatment (Obs), and 5 rabbits received 2 mg/kg/day aspirin (Obs+aspirin), administered by an osmotic pump …

Malemedicine.medical_specialtyUrologyInjections SubcutaneousUrinary BladderUrologyConnective tissueStimulationurologic and male genital diseasesBladder outlet obstructionBleeding timeInternal medicinemedicineAnimalsDrug ImplantsAspirinmedicine.diagnostic_testAspirinbusiness.industryMuscle SmoothOrgan SizeUrinary Bladder Neck ObstructionNeck of urinary bladderEndocrinologymedicine.anatomical_structurePlatelet aggregation inhibitorRabbitsbusinessPerfusionmedicine.drugUrology
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�ber die Rolle des Histamins bei der Verk�rzung der Blutungszeit nach Adrenalin

1955

Die Verkurzung der Blutungszeit nach Adrenalin konnte durch die Antihistaminica Casantin oder Luvistin sowohl aufgehoben als auch verhindert werden. Die Versuche sprechen dafur, das Histamin an der Verkurzung der Blutungszeit nach Adrenalin wesentlich beteiligt ist.

Pharmacologychemistry.chemical_compoundHistamine Effectsmedicine.diagnostic_testchemistryBleeding timebusiness.industryPharmacology toxicologymedicineGeneral MedicinePharmacologybusinessHistamineNaunyn-Schmiedebergs Archiv f�r Experimentelle Pathologie und Pharmakologie
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